S-equol Modulates T3-Induced Transcription and Neurite Outgrowth in Neuronal Cells.
📄 Abstract
Thyroid hormones (THs) and estrogen (E2) play essential roles in neuronal differentiation and plasticity during brain development. S-equol, a plant-derived isoflavone metabolite, is a selective E2 receptor (ER) ligand that exhibits neurotrophic effects; however, its interaction with TH receptor (TR) signaling remains unclear. In this study, we investigated the effects of S-equol on TR
Confidence:
0.03
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Идентификация (6 полей)
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